ATLS EXAM WITH COMPLETE SOLUTIONS
QUESTIONS AND ANSWERS GRADED A
True or false? Although the mechanism of injury may be similar to those for the
... [Show More] younger population,
data shows increased mortality with similar severity of injury in older adults.
ANS: True
In the elderly population, what is decreased physiological reserve?
ANS: aging is characterized by impaired adaptive and homeostatic mechanisms that caused an
increased susceptibility to the stress of injury. Insults tolerated by the younger population can lead to
devastating results in elderly patients.
Pre-existing conditions that affect morbidity and mortality include:
ANS: cirrhosis, coagulopathy, COPD, ischemic heart disease, DM
What is the most common mechanism of injury in the elderly?
ANS: Fall. Nonfatal falls are common in women and fractures are common in women who fall. Falls
are the most common cause of TBI.
In the elderly population, what are risk factors for falls?
ANS: advanced age, physical impairment, history of previous fall, medication use, dementia,
unsteady gait, and visual, cognitive impairment
Most of elderly traffic fatalities occur in the daytime and on weekends and typically involve other
vehicles. Why?
ANS: Older people drive on more familiar roads and at lower speeds and tend to drive during the day.
Older people have slower reaction time, a larger blind spot, limited cervical mobility, decreased
hearing, and cognitive impairment.
True or False? Mortality associated with small to moderate sized burns in older adults remains high
ANS: True
Spilled hot liquids on the leg, which in younger patients may re-epithelialize due to an adequate
number of hair follicles, will result in a full thickness burn in older patients.
ANS: this is true
Airway-patients may have dentures that may loosen or obstruct the airway. If dentures are not
obstructing the airway, leave them in place for what?
ANS: bag mask ventilation, as it improves mask fitting.
When preforming rapid sequence intubation, the dose of benzos, barbiturates, and other sedatives
should be reduced to what percentage to minimize the risk of cardiovascular depression?
ANS: 20-40%
Functional changes in cardiac system include declining function, decreased sensitivity to catechol
amines, atherosclerosis of coronary vessels, increased afterload, fixed heart rate (beta blockers)
ANS: this results in lack of classic response to hypovolemia, risk for cardiac ischemia, elevated BP at
baseline, and increased risk of dysrhythmias.
Functional changes in pulmonary system include decreased elastic recoil, reduced residual capacity,
decreased gas exchange and decreased cough reflex
ANS: thus they are at increased risk for respiratory failure, increased risk for pneumonia, and poor
tolerance to rib fractures
Functional changes in renal system include loss of renal mass, decreased GFR, and decreased
sensitivity to ADH and aldosterone
ANS: resulting in drug dosing for renal insufficiency, decreased ability to concentrate urine, increased
risk for AKI and urine flow may be normal with hypovolemia
Functional changes to MSK include loss of lean body mass, osteoporosis, changes in joints and
cartilage, c spine degenerative changes and loss of skin elastin and subcutaneous fat
ANS: resulting in increased risk for fractures, decreased mobility, difficulty for oral intubation, risk of
skin injury, increased risk for hypothermia, challenges in rehabilitation
Functional changes in Endocrine system include decreased production and response to thyroxin and
decreased dehydroepiandrosterone (DHEA)
ANS: resulting in occult hypothyroidism, relative hyper cortisone states and increased risk of infection
True or false: Arthritis can complicate the airway and cervical spine. Patients can have multilevel
degenerative changes affecting disk spaces and posterior elements associated with severe central
canal stenosis, cord compression, and myelomalacia
ANS: true
In elderly population, due to their changes in pulmonary system, placing a gauze between gums and
cheek to achieve seal when using bag valve mask ventilation is okay. In addition, because aging
causes a suppressed heart rate response to hypoxia......
ANS: respiratory failure may present insidiously in older adults.
Age related changes in the cardiovascular system place the elderly trauma patient at significant risk
for being inaccurately categorized as hemodynamically stable.
ANS: Elderly patients have a fixed heart rate and fixed cardiac output, thus, their response to
hypovolemia will involve increasing their systemic vascular resistance. Furthermore, since older
patients have HTN, an acceptable BP may truly reflect a hypotensive state. A systolic BP of 110 is to
be utilized as the threshold for identifying hypotension in patients 65 and older.
Do no equate blood pressure with shock in older patients
ANS: BP in older patients may look normal due to the medications they are on. Use lactate and base
deficit to evaluate for evidence of shock
what 2 factors place elderly patients at risk for intracranial hemorrhage?
ANS: aging causes dura to become more adherent to the skull increasing risk of injury and older
patients are on anticoagulant and antiplatelet medications.
Loss of subcutaneous fat, nutritional deficiencies, chronic medical conditions place elderly patients as
risk for hypothermia and complications for immobility.
ANS: Rapid evaluation and when possible early liberation from spine boards and cervical collars will
minimize complications.
True or False: Fall prevention is the mainstay of reducing the mortality associated with pelvic
fractures.
ANS: true
poor hygiene, dehydration, oral injury, contusions affecting the inner arms, inner thighs, palms, soles,
scalp, ear, nasal bridge and temple injury from being struck while wearing glasses, contact burns and
scalds. These are all signs of.......?
ANS: Elder maltreatment. The presence of physical findings of maltreatment should prompt a detailed
history. if history conflicts with findings, immediately report findings to authorities.
True of false: early activation of the trauma team may be required for elderly patients who do not
meet traditional criteria for activation
ANS: True. A simple injury such as an open tibia fracture in a frail elderly patient may become life
threatening.
Common mechanisms of injury include falls, MVC, burns, and penetrating injuries
ANS: common injuries in the elderly include rib fractures, TBI, pelvic fractures
The best initial treatment for the fetus is to provide optimal resuscitation of the mother. True or False?
ANS: True. Also if xray examination is indicated during the pregnant patient's treatment, it should not
be withheld because of the pregnancy.
What happens as the uterus enlarged and the bowel is pushed cephalad.
ANS: When the uterus enlarges it pushes the bowel cephalad and the uterus lies in the upper
abdomen. As a result, the bowel is somewhat protected from blunt abdominal trauma, whereas the
uterus and its contents (fetus and placenta) become more vulnerable. Uterus remains intrapelvic until
12 weeks and then at 20 weeks it is at the umbilicus, and at 34-36 weeks it reaches the costal
margin.
Amniotic fluid can cause amniotic fluid embolism and disseminated intravascular coagulation
following trauma if fluid enters maternal intravascular space. True or False
ANS: True
By the third trimester, what is the complication of trauma to the pelvis of the mother?
ANS: by the third trimester, the uterus is large and thin walled. In vertex presentation, fetal head is
usually in the pelvis and the remainder of the fetus is exposed above the pelvic brim. Pelvic fractures
in late gestation can result in skull fracture or intracranial injury to the fetus. Also we can have a
placental abruption due to its little elasticity and vulnerability to sheer forces.
An abrupt decrease in maternal intravascular volume can result in a profound increase in uterine
vascular resistance reducing fetal oxygenation despite reasonably normal maternal vital signs.
ANS: this is true
Physiological anemia of pregnancy
ANS: A smaller increase in red blood cell volume can occur resulting in a decreased hematocrit level.
Thus, in late pregnancy a hematocrit of 31-33% is normal.
Healthy pregnancy patients can lose 1200-1500 mL of blood before exhibiting signs and symptoms of
hypovolemia. How can this manifest?
ANS: this amount of hemorrhage may be reflected by fetal distress as evidenced by an abnormal
fetal heart rate.
What are some of the lab changes in pregnancy?
ANS: WBC increases to 12000 and during labor can be 25000. Fibrinogen and other clotting factors
are mildly elevated and PT and pTT are shortened, but bleeding time and clotting time are
unchanged.
After the 10th week of pregnancy, cardiac output can increase 1.0-1.5 L/min because of the increase
in plasma volume and decrease in vascular resistance of the uterus and placenta.
ANS: The placenta receives 20% of the patient's cardiac output during the 3rd trimester. In supine
position, vena cava compression can decrease cardiac output by 30% because of decreased venous
return from lower extremities.
During pregnancy the heart rate increases to a maximum of 10-15 beats per minute over baseline by
the third trimester.
ANS: this heart rate must be considered when interpreting a tachycardia response to hypovolemia.
Blood pressure falls 5-15 mm Hg in systolic and diastolic pressures during second trimester, although
it returns to near normal levels at term.
ANS: some women experience hypotension when placed in the supine position due to the
compression of teh inferior vena cava.
hypertension in the pregnant if accompanied by proteinuria may represent what?
ANS: pre-eclampsia.
EKG findings in pregnant patient
ANS: Flatted or inverted T waves in leads III and AVF and the precordial leads may be normal.
Ectopic beats are increased during pregnancy.
Minute ventilation increases primarily due to an increase in tidal volume. Hypercapnia (30 mm Hg) is
common in late pregnancy
ANS: Monitor ventilation in late pregnancy with arterial blood gas values. A PaCO2 of 35-40 mm Hg
may indicate impending respiratory failure during pregnancy. Pregnant patients should be hypo
apneic.
Anatomical alterations in the thoracic cavity seem to account for the decreased residual volume
associated with diaphragmatic elevation and chest x ray reveals increased lung marking and
prominence of the pulmonary vessels.
ANS: oxygen consumption increases during pregnancy and its important when resuscitating injured
pregnant patients to maintain adequate oxygenation above 95%
In patients with advanced pregnancy, those that require a chest tube placement, where should the
test tube be placed?
ANS: it should be positioned higher to avoid intra-abdominal placement given the elevation of the
diaphragm.
Urinary system: what happens to the GFR, serum creatinine and urea nitrogen levels?
ANS: GFR and renal blood increases during pregnancy, whereas levels of the serum creatinine and
urea nitrogen fall to one half of the normal pre pregnancy levels. Glycosuria is common in pregnancy.
When interpreting x ray films of the pelvis in a pregnant patient, the symphysis pubis widens 4-8 mm
and the sacroiliac joint spaces increase by the 7th month
ANS: keep this in mind
Eclampsia
ANS: Maintain a high index of suspicion for eclampsia when seizures are accompanied by HTN,
proteinuria, hyperreflexia, and peripheral edema in pregnant trauma patients. This can mimic head
injury.
External contusions and abrasions of the abdominal wall are signs of blunt uterine trauma.
ANS: true. Fetal injuries can occur when the abdominal wall strikes an object, such as the dashboard
or steering wheel, or when a pregnant patient is struck by a blunt instrument.
Using a shoulder restraints in conjunction with a lap belt reduces the likelihood of direct and indirect
fetal injury, presumably because the shoulder belt dissipates deceleration forces over a great surface
area and helps prevent the mother from flexing forward over the gravid uterus.
ANS: the deployment of air bags in vehicles does not appear to increase pregnancy specific risks.
Using lap belt alone allows for forward flexion and uterine compression with possible uterine rupture
or placental abruption. Lap belt worn too high over uterus may produce uterine rupture.
Penetrating injury to pregnant women
ANS: As uterus grows larger, other viscera are protected from penetrating injury. Dense uterine
musculature in early pregnancy can absorb significant amount of energy from penetrating objects
decreasing their velocity and lowering risk of injury to other viscera. However, fetal outcome is
generally poor with penetrating injury to uterus.
carefully observe pregnant patients with even minor injuries since occasionally minor injuries are
associated with placental abruption and fetal loss.
ANS: True. AND to optimize outcomes for mother and baby, clinicians must assess and resuscitate
the mother first and then assess the fetus before conducting second survey of the mother.
Failure to displace the uterus to the left side in a hypotensive pregnant patient
ANS: logroll all patients appearing clinically pregnant (second and third trimester) to the left 15-30
degrees and elevate the right side 4-6 inches and support with a bolstering device to maintain spinal
motion restriction and decompression of the vena cava.
Due to increases intravascular volume, pregnant patients can lose a significant amount of blood
before tachycardia, hypotension, and other signs of hypovolemia occur. Thus, what do stable vital
signs in a pregnant patient indicate about the fetus?
ANS: The fetus may be in distress and the placenta deprived of vital perfusion while the mother's
condition and vital signs appear stable. Administer crystalloid fluid resuscitation and blood to support
the physiological hypervolemia of pregnancy. vasopressers should be an absolute last resort in
restoring maternal blood pressure as they further reduce uterine blood flow, resulting in fetal hypoxia.
What does a normal fibrinogen level indicate in a pregnant patient?
ANS: Fibrinogen level may double in late pregnancy and a normal level may indicate early
disseminated intravascular coagulation
Most common cause of fetal death?
ANS: maternal shock and maternal death. Placental abruption is second. Placental abruption is
suggested by vaginal bleeding, uterine tenderness, frequent uterine contractions, uterine tetany, and
uterine irritability (uterus contracts when touched). In 30% of cases of abruption, bleeding may not
occur. Uterine ultrasound may be helpful in diagnosis, but is NOT definitive.
Signs of uterine rupture
ANS: abdominal tenderness, guarding, rigidity, or rebound tenderness. Signs of peritonitis are hard to
tell due to expansion and attenuation of the abdominal wall musculature. Other findings include
abdominal fetal lie (oblique or transverse lie), easy palpation of the fetal parts because of their
extrauterine location and inability to readily palpate the uterine fundus when there is fundal rupture.
Xray evidence of rupture include extended fetal extremities, abnormal fetal position, and free
intraperitoneal air.
Perform continuous fetal monitoring with a tocodynamometer beyond 20-24 weeks of gestation.
ANS: Patients with no risk factors for fetal loss should have continuous monitoring for 6 hours,
whereas, patients with risk factors for fetal loss or placental abruption should be monitored for 24
hours. RISK FACTORS ARE: heart rate > 110, an injury severity score >9, evidence of placental
abruption, fetal heart rate >160 or less than 120, ejection during MV, and motorcycle or pedestrian
collisions
REMEMBER: maternal bicarbonate is low during pregnancy to compensate for respiratory alkalosis.
ANS: 17-22 in pregnant patient. (non pregnant patient is 22-28)
Fetal heart rate is a sensitive indicator of maternal blood volume status and fetal well being.
ANS: normal range for fetus is 120-160. abnormal heart rate, repetitive decelerations, absence of
accelerations or beat to beat variability and frequent uterine activity can be signs of impending
maternal and or fetal decompensation (hypoxia or acidosis) and should prompt immediate obstetrical
consultation.
If a DPL is to be placed in a pregnant trauma patient, place the catheter above the umbilicus using
the open technique. Be alert to uterine contractions which suggest early labor and tetanic
contractions which suggest placental abruption.
ANS: evidence of ruptured chorioamniotic membranes include amniotic fluid in vagina evidenced by a
pH of 4.5
Bleeding in 3rd trimester may indicate placental abruption and impending death of the fetus, a vaginal
exam is vital
ANS: however, avoid repeating vaginal examination, CT abdominal imaging can be done and
radiation doses less than 50mGy are not associated with fetal anomalies or higher risk of fetal loss.
Admission to hospital for pregnant patients:
ANS: vaginal bleeding, uterine irritability, abdominal tenderness, pain or cramping, evidence of
hypovolemia, changes in or absence of fetal heart tones and or leakage of amniotic fluid
With extensive placental separation or amniotic fluid embolization, widespread consumptive
coagulopathy can emerge rapidly causing depletion of fibrinogen, other clotting factors, and platelets.
ANS: immediately perform uterine evacuation and replace platelets, fibrinogen, and other clotting
factors.
As little as 0.01mL of RH+ blood will sensitize 70% of Rh- women.
ANS: All pregnany RH negative trauma patients should receive RH immunoglobulin therapy unless
injury is remote from the uterus (isolated distal extremity injury)
Intimate partner violence in pregnant patient:
ANS: injuries inconsistent with history, diminished self image, depression or suicide attempts, self
abuse, frequent ED visits, symptoms suggestive of substance abuse, isolated injuries to the gravid
abdomen, parter insists on being present for the interview and exam and monopolizes discussion
What is the difference between burns and other injuries?
ANS: The biggest difference is that the consequences of burn injury are directly linked to the extent of
the inflammatory response to the injury. The larger and deeper the burn, the worse the inflammation.
Flame injury is more evident than most chemical injuries.
ANS: Monitor IV lines closely to ensure they do not become dislodged as the patient becomes more
edematous. Regularly check ties securing ET or NG to ensure they are not too tight.
Factors that increase the risk of upper airway obstruction are:
ANS: increasing burn size and depth, burns to the head and face, inhalation injury, associated
trauma, and burns inside the mouth. Airway can become obstructed form direct injury such as
inhalation injury, but also from massive edema resulting from burn injury.
How do you decontaminate burn areas?
ANS: Completely remove the patient's clothing to stop burning process, but do not peel off adherent
clothing. Synthetic fabrics can ignite, burn rapidly at high temps and melt into hot residue that
continues to burn the patient. brush any dry chemical powder from wound. rinse with copious
amounts of warm saline irrigation or rinsing in a warm shower. once the burning process has been
stopped, cover the patient with warm, clean, dry linens to prevent hypothermia.
hoarseness, stridor, accessory respiratory muscle use, sternal retraction are signs of what?
ANS: airway obstruction. Clinical manifestations of inhalation injury may be subtle and may not show
up within the first 24 hours. do not wait for the xray to show evidence of pulmonary injury or changes
in blood gas because airway edema can preclude intubation and a surgical airway will be required.
A carboxyhemoglobin level greater than what percentage indicates a patient was involved in a fire
and has inhalation injury?
ANS: 10%
Indications for early intubation in burn patients:
ANS: full thickness circumferential neck burns, signs of airway obstruction, extent of the burn > 40%,
burns inside the mouth, difficulty clearing secretions or swallowing, decreased level of
consciousness,
Patient with inhalation injury are at risk for bronchial obstruction from secretions and debris and they
may require bronchoscopy.
ANS: Make sure to place an adequately sized airway tube
Direct thermal injury to the lower airway is very rare and essentially occurs only after exposure to
superheated steam or ignition of inhaled inflammable gases. Breathing concerns arise from what 3
general causes:
ANS: hypoxia, carbon monoxide poisoning, and smoke inhalation injury.
Always assume CO exposure in patients who were burned in enclosed areas. Patients with CO levels
less than 20% may not show any symptoms
ANS: HA and nausea (20-30%), confusion (30-40%), coma (40-60%) and death (>60%). Cherry red
skin color in patients may only be seen in moribund patients.
Measurements of arterial PaO2 do not reliably predict CO poisoning b/c a partial pressure of only 1
mm Hg results in an HbCO level of 40% or greater. Pulse ox cannot be relied on to rule out carbon
monoxide poisoning b/c we cant distinguish oxyhemoglobin from carboxyhemoglobin. A discrepancy
between pulse ox and arterial blood gas may be explained by presence of carboxyhemoglobin.
Cyanide inhalation poisoning can occur in confined spaces and sign of potential toxicity is persistent
profound unexplained metabolic acidosis. ANS: THERE IS NO ROLE for hyperbaric oxygen therapy
in the primary resuscitation of a patient with critical burn injury.
American Burn Association states 2 requirements for diagnosis of smoke inhalation injury:
ANS: 1. exposure to combustible agent
2. signs of exposure to smoke in the lower airway, below the vocal cords, seen on bronchoscopy.
A chest Xray and arterial blood gases should be ordered to evaluate the pulmonary status of a patient
with smoke inhalation injury, but normal values on admission DO NOT exclude an inhalation injury.
The treatment of smoke inhalation injury is supportive.
ANS: Any patient with smoke inhalation injury and significant burns greater than 20% TBSA should
be intubated. IF the patient's hemodynamic condition permits and spinal injury has been excluded,
elevate the patient's head and chest 30 degrees to help reduce neck and chest wall edema.
True or false: Clinicians should provide burn resuscitation fluids for deep partial and full thickness
burns larger than 20% TBSA
ANS: True. urine output monitoring is 0.5mL/kg/hr in adults and should be maintained at 30-50cc/hr
to minimize over resuscitation
in a burn patient, cardiac dysrhythmias may be the first sign of hypoxia and electrolyte or acid base
abnormalities.
ANS: therefore an ECG should be performed for cardiac rhythm disturbances. Persistent academia in
patients with burn injuries may be due to under resuscitation or infusion of large volumes of saline.
Tachycardia is a poor indication for resuscitation in the burn patient.
ANS: Adjust the fluid rate up or down based on the urine output and recognize that factors such as
inhalation injury, age of patient, renal failure, diuretics, and alcohol can affect the volume of
resuscitation and urine output.
True of false: Burn patients should get tetanus.
ANS: true
Partial thickness burns
ANS: are characterized as either superficial partial thickness (moist, painfully hypersensitive, ,
potentially blistered, homogenously pink, and blanch to touch) or deep partial thickness ( drier, less
painful, potentially blistered, red or mottled in appearance, and do not blanch to touch)
Full thickness burns
ANS: appear leathery and skin may be white or translucent or waxy white. surface area is painless to
light touch or pinprick and generally dry
Compartment syndrome in burn patients:
ANS: Compartment syndrome can result from an increase in pressure inside the compartment that
interferes with perfusion to the structures within that compartment. In burns, this condition results
from a combination of decreased skin elasticity and increased edema in the soft tissue. A pressure >
30 mm Hg within the compartment can lead to muscle necrosis and once the pulse is gone it may be
TOO LATE to save the muscle. so recognize the signs early:
pain greater than expected and out of proportion to the injury
pain on passive stretch of the affected muscle
tense swelling of the affected compartment
paresthesias or altered sensation distal to the affected compartment
compartment syndrome may be present with circumferential chest and abdominal burns
ANS: chest and abdominal escharotomies performed along the anterior axillary lines with cross
incision at the clavicular line and the junction of the thorax and abdomen usually relieve this problem.
relieve circulatory compromise in a circumferentially burned limb by eschartomy and these
escharotimies are not needed within the first 6 hours.
Partial thickness burns are painful when air currents pass over the burned surface.
ANS: gently cover the burn with clean sheets will decrease the pain and deflect air currents. Do not
break blisters or apply an antiseptic and application of cold compress can cause hypothermia. DO not
apply cold water to a burn patient.
electrical burns are more serious than they appear on the body surface and extremities, particularly
the digits.
ANS: current can travel inside blood vessels and nerves and can cause local thrombosis and nerve
injury. So a severe electrical injury usually results in contracture of the affected extremity.
A clenched hand with a small electrical entrance wound should alert the clinician that a deep soft
tissue injury is likely much more extensive than is visable to the naked eye
ANS: true. patients with severe electrical injuries require fasciotomies. Electricity can cause forced
contraction of muscles, doctors need to examine patient for skeletal and muscular damage,
especially for fractures of the spine and rhabdomyolysis
How do you dissolve a tar burn?
ANS: Use mineral oil
Abuse and burns
ANS: circular burns and burns with clear edges and unique patterns may reflect cigarette burns or
iron. Burns on the sole of the feet usually suggest child was placed in hot water. A burn on the
posterior aspect of the LE and buttocks
Patient with electrical burn can develop for acute renal failure
ANS: remember these burns can cause serious muscle damage without showing signs outright. Test
urine for hem chromogen and administer proper volume. Assess for compartment syndrome and
attach EKG leads as electrical injury can cause arrhythmias.
Frostbite is due to freezing of tissue with intracellular ice crystal formation, microvascular occlusion,
and subsequent tissue anoxia.
ANS: first degree: hyperemia and edema are present w/o skin necrosis second degree: large clear
vesicles accompany the hyperemia and edema with partial thickness skin necrosis. third degree
frostbite: full thickness skin necrosis including muscle and bone with later necrosis
treatment is circulating water at constant 40 degrees C or 104F until pink color and perfusion return
in 20-30 minutes.
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