Diagnosis Study Guide for Advanced
Pathopharmocology
Advanced Pathopharmacological Foundations
(Western Governors University)
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Diagnosis Study Guide
In addition to pharmacology knowledge, mastery of pathophysiology knowledge is required for this course. The
concept map below outlines key components to focus on when studying.
Subjective Patient History (Chief Complaint, History of Present Illness, Past Medical/Surgical/Family
History) – What symptoms would you expect the patient to report? Are there concomitant diagnoses that occur in
patients with the condition? Is there a genetic component found with the condition? Race, ethnic or gender
commonalities with the condition? Social history such as tobacco, alcohol or illicit drug use preceding in those with the
condition?
Objective Patient Findings (Height, weight, vital signs and physical exam) – Pertinent positives and pertinent
negative findings you would expect in a patient with the condition.
Diagnostics (labs, radiology, exam findings, electrophysiology testing): What are the best practice
recommendations for diagnosing the condition? Is there an algorithm or tiered approach when attempting to diagnose
the condition? Are there screening tools recommended? Likelihood of false positives or negatives? How reliable is the
approach in diagnosing with certainty?
Non-pharmacological Management: What are lifestyle modifications used to treat the condition?
Pharmacologic Management: What are the first line, second line and/or common medications used to manage this
condition?
Primary
Hypertension
Resistant
Hypertension
PTSD Diabetes Alzheimer’s Disease
Celiac Disease Crohn’s Disease Social Determinants of
Health
POTS Chronic Obstructive
Pulmonary Disease
Myasthenia
Gravis
Hashimoto’s
Thyroiditis
Hypothyroidism Small Cell Lung
Carcinoma
Adult/Pregnant
Urinary Tract
Infection
Thyroid Storm Ulcerative Colitis Medullary Thyroid
Cancer
Generalized Anxiety
Disorder
Community
Acquired Pneumonia
Neuropathic Pain G6PD Deficiency Tachydysrhythmias Schizophrenia Cancer
Nephritis Depression Heart Disease Influenza Group A strep
pharyngitis
Pleural Effusion Chronic joint pain Parathyroid function Kidney physiology &
regulation of blood
pressure
Tuberculosis – PPD
low, high risk
patients
Small cell lung Thalassemias Color blindness
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carcinoma
Conditions of
Pregnancy
Use of live
vaccinations
(varicella, MMR)
Urinary Tract Infection Varicella exposure Breastfeeding & oral
contraceptives
Pediatric
Conditions
Urinary Tract
Infection
Otitis media Ataxia Asthma Color blindness
Bradycardia
Sports Physical
Sickle Cell Crisis Thrush/Diaper
Candidiasis
Fragile X Fragile X
Hypoplastic Left
Heart Syndrome
Sickle cell
prophylaxis
strategies of the
newborn
Chickenpox – pain
management
modalities
Hutchinson-Gilford
Progeria Syndrome
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Pharmacologic Management
Can be caused by genetic factors
Inc. Sodium intake
Dec. physical activity,
psychosocial stress, and obesity
Primary has no known cause
(genetics and environment
R.F= smoking, age, obesity,
Stage 1: 130-139 or 80-89
Stage 2: >140 or >90
HTN Crisis: >180 or >120
“Silent disease”
Commonly recommended
medications include thiazide
diuretics, ACE inhibitors or
ARBs, and calcium channel
blockers.
Inc. in Cardiac output,
total peripheral
resistance, or both
Physical training increases stroke
volume, which has the effect of
lowering heart rate and hence
systolic blood pressure, and should
consist of regular aerobic physical
>130 or >80
Diagnosis requires the measurement of
blood pressure on at least two separate
occasions averaging two readings at least
2 minutes apart, with the individual
seated, the arm supported at heart level,
after 5 minutes rest, with no smoking or
caffeine intake in the past 30 minutes.
activity. Individuals Daorwenlcoaoduednbsyeslieerdrattaongo1 ([email protected]) iagnostic tests include complete blood
Diagnostic methods
Primary HTN
Objective Patient Subjective Patient
Non-Pharmacologic
Management Strategies
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Resistant HTN
Objective Patient Subjective Patient
Pharmacologic Management
Diagnostic methods
Non-Pharmacologic
Management Strategies
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Symptoms must last more than a
month and be severe enough to
interfere with relationships or work to
be considered PTSD.
antidepressants
Psychotherapy
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Objective Patient Subjective Patient
PTSD
Pharmacologic Management
Diagnostic methods
Non-Pharmacologic
Management Strategies
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Type 1:= insulin deficient
(dependent)
Type 2:= insulin resistant
FPG 100 to 125 mg/dL 2. 2-hr PG in
the range of 140 to 199 mg/dL
during an OGTT 3. HbA1c 5.7% to
6.4%
The most well-recognized risk
Obesity
Polyuria, polydipsia, polyphagia, weight
loss, fatigue
Type 2: fatigue, pruritus, recurrent
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